From your link:

mRNA-1273 booster vaccination-associated elevation of markers of myocardial injury occurred in about one out of 35 persons(2.8%), a greater incidence than estimated in meta-analyses of hospitalized cases with myocarditis (estimated incidence 0.0035%) after the second vaccination.14,15 Elevated hs-cTnT was independent of previous COVID infection or the interval since the last vaccine dose. Among the overall group of participants, hs-cTnT concentration on day 3 after mRNA-1273 booster vaccination as a continuous variable, was significantly higher compared to a well-matched control cohort. Second, all cases were mild with only a transient and short period of myocardial injury (maximum hs-cTnT concentration 35ng/L).

It goes on to say:

Thus, for the majority of individuals, the overall very favorable risk-benefit ratio of booster immunizations persists.

The concluding paragraph:

In conclusion, using active surveillance, mRNA-1273 vaccine-associated mild transient myocardial injury was found to be much more common than previously thought. It occurred in one out of 35 persons, was mild and transient, and more frequent in women versus men. Neither anti-IL-1RA, nor pre-existing vaccine/infection-induced immunity or systemic inflammation seemed to be dominant mechanisms of myocardial injury. No participant developed MACE within 30-day

MACE stands for “Major adverse cardiac events”

I’m not sure what your angle is/was, but the study sounds like it’s not really a big deal at all, compared to the adverse effects of getting COVID without the jab. It’s only interesting from a medical study standpoint.